According to a new study, the neural and cognitive complexity of the octopus could originate
from a molecular analogy with the human brain.
New research has identified an important molecular analogy that could explain the remarkable intelligence of these fascinating invertebrates.
An exceptional organism with an extremely complex brain and cognitive abilities makes the octopus very unique among invertebrates. So much so that it resembles vertebrates more than invertebrates in several aspects.
The neural and cognitive complexity of these animals could originate from a molecular analogy with the human brain, as discovered by a research paper that was recently published in BMC Biology and coordinated by Remo Sanges from Scuola Internazionale Superiore di Studi Avanzati (SISSA) of Trieste and by Graziano Fiorito from Stazione Zoologica Anton Dohrn of Naples.
This research shows that the same ‘jumping genes’ are active both in the human brain and in the brain of two species, Octopus vulgaris, the common octopus, and Octopus bimaculoides, the Californian octopus.
This research shows that the same ‘jumping genes’ are active both in the human brain and in the brain of two species, Octopus vulgaris, the common octopus, and Octopus bimaculoides, the Californian octopus.
A discovery that could help us understand the secret of the intelligence of these remarkable organisms.
Sequencing the human genome revealed as early as 2001 that over 45% of it is composed of sequences called transposons, so-called ‘jumping genes’ that, through molecular copy-and-paste or cut-and-paste mechanisms, can ‘move’ from one point to another of an individual’s genome, shuffling or duplicating.
In most cases, these mobile elements remain silent: they have no visible effects and have lost their ability to move. Some are inactive because they have, over generations, accumulated mutations; others are intact, but blocked by cellular defense mechanisms.
Sequencing the human genome revealed as early as 2001 that over 45% of it is composed of sequences called transposons, so-called ‘jumping genes’ that, through molecular copy-and-paste or cut-and-paste mechanisms, can ‘move’ from one point to another of an individual’s genome, shuffling or duplicating.
In most cases, these mobile elements remain silent: they have no visible effects and have lost their ability to move. Some are inactive because they have, over generations, accumulated mutations; others are intact, but blocked by cellular defense mechanisms.
From an evolutionary point of view even these fragments and broken copies of transposons can still be useful, as ‘raw matter’ that evolution can sculpt. READ MORE...
